More than two years after the coronavirus pandemic began, researchers are still struggling to understand how the virus causes the onset of dementia.
In many ways, the virus has been the biggest mystery.
But it is the brain disease that has made it so hard for scientists to figure out what it is and how it affects people.
And it has been a problem that has continued to worsen since the coronavectirus pandemics first hit.
The brains of people with dementia have the same structure and functions as the brains of healthy people.
The brain does not need to be constantly pumping blood, and blood flow is unaffected by brain swelling.
But the brain is a delicate organ that requires constant monitoring, so its electrical activity is not always predictable.
This has meant scientists have to rely on a variety of techniques to figure this out.
“We’ve got a whole range of things that are being tested for, so we’re working on these things,” says Dr Steven Mearns, a neuroscientist at the University of Queensland’s Brain and Mind Institute in Brisbane.
In the past, these tests have been quite limited, so many researchers have looked at how brain activity is affected by oxygen levels, whether a person has a brain tumor or a damaged hippocampus, and whether the brain has damaged its outer membrane, which is responsible for blood flow.
They’ve been unable to figure how the brain’s electrical activity changes in response to changes in oxygen levels.
That’s where the term “brain abscess” comes from.
It refers to a type of infection that is found in the brain, and is usually a result of damage to one or more brain cells, such as the ones that process information.
But over time, the cells can become damaged beyond repair, leading to inflammation and damage to the brain.
The result is inflammation, swelling, and damage.
“That is a real challenge because we’re always trying to get the signal to the cells to make them go into damage mode,” Dr Mearnes says.
“When we have that, we’re looking at all the signals that are going out of the cells and that can cause the abscess.”
This is what the term brain absence means.
Brain abscess is a condition that occurs when the brain cells that process signals can’t properly heal.
It may also occur after surgery or an accident.
When this happens, the brain loses some of its ability to communicate with other parts of the brain and may not function normally.
But if it happens too often, it can become very serious.
Dr Mowrey says this can lead to a brain infection that can lead in the wrong direction.
It can also lead to brain damage, or damage to areas of the body that need protection from the virus.
There is no known cure for brain absences, but there are treatments that have been shown to help people with the virus manage their symptoms.
One of these is neuro-inflammation.
Neuro-inflammations occur when cells of the immune system are damaged.
It’s a disease of inflammation that is linked to many neurological conditions.
For example, an increase in inflammatory responses can lead people with Alzheimer’s disease to become more vulnerable to infection, and can also cause an increase or even a decline in certain types of memory, attention and other cognitive skills.
In some cases, this leads to memory loss, confusion, and other problems.
Dr Thomas Wetherington is a researcher at the Brain and Memory Institute at Queensland University of Technology.
He says it is important to look at different types of brain absents and to understand where they come from.
“I think it’s important to recognise that brain absense is a problem in many different brain diseases and different conditions,” he says.
For people with mild cognitive impairment, for example, there may be no brain absessions in the first place.
But when it comes to more severe conditions, such a condition can develop and lead to the loss of some of the basic functions of the human brain.
“There is a really large number of brain disorders, and there’s a lot of work that’s still being done in these areas to understand the impact of the virus on different brain disorders,” he adds.
“For example, we know that the virus does not change the brain structures of the cortex or the hippocampus.”
It’s not clear exactly how much damage occurs to brain cells in mild cognitive impairments, but Dr Wetherton says it can be significant.
“People who are not impaired in terms of cognitive function are more susceptible to this infection, because it’s easier to infect them than to treat them,” he explains.
“It’s a more serious problem, because we know it’s much harder to treat.”
But in mild impairment, it may be more difficult to detect this type of damage, and if it occurs, it is much less likely to cause permanent damage to brain tissue.
“In mild impairment where the brain does have normal function,